Short-term exposure to side effects is low; long-term high levels of resistance to rheumatism have adverse effects。
The gastrointestinal reaction is most common. Tasting directly stimulates stomach mucous membranes, causing abdominal discomfort, nausea and vomiting. High blood concentrations stimulate the ctz and can cause nausea and vomiting. Larger doses of oral treatment (anti-typhoid treatment) can cause stomach ulcer and undetectable stomach haemorrhage (free from pain); those with pre-existing ulcer diseases have increased symptoms. After eating, take drugs, chew tablets, and co-opt acids such as calcium carbonate, or take intestines to mitigate or avoid the above reactions. The internal source pg has a protective effect on the gastric mucous membrane, which can reduce stomach haemorrhage caused by pge2 by co-combining it with acetyl acrylic acne, and is proportional to the dose of pge2, suggesting that the acetyl acid ulcers may be associated with its inhibition of gastric mucous membrane synthesis. The stomach ulcer is banned。
The general dose of acetyl acid for coagulation disorders inhibits the accumulation of platelets and prolongs bleeding. Large doses (above 5g/day) or long-term administration can also inhibit the formation of a condensed enzymes, prolonging the enzymes and preventing vitamin k. Severe liver damage, low-condensation enzymes, vitamin k deficiency should be avoided. The first week of the operation should be discontinued。
3. A small number of patients with an allergy reaction can experience measles, angiogenesis and an allergic shock. Some asthma patients can induce asthma on acetyl acid or other antithermal analgesics, called “aspirin asthma”, which is not an allergic reaction based on antigen-antibodies but is related to their inhibition of pg biosynthesis. As a result of the obstruction of the synthesis of the pg, the increase in the production of white triene and other lipidase metabolites from peanut tetraolec acid and the dominance of endogenous bronchial constrictors have led to bronchial convulsions that induce asthma. Adrenalin treatment “aspirin asthma” is ineffective. Acetylic acid is banned for asthma, nasal meat and chronic measles patients。
4. Hydraulic acid response 4. Acetyl acid reaction. 5 g/day, headaches, dizziness, nausea, vomiting, ringing in the ear, sight, hearing loss, commonly referred to as aqueous acid reaction, are a manifestation of aqueous acid poisoning. Severely severe cases can result in over-respiration, alkalin balance disorder or even mental disorders. The severely intoxicated person should immediately stop the drug and enter the sodium carbonate solution with an alkaline urine to accelerate the discharge of the aqueous acid。
5. The reye syndrome reported a risk of abrasive syndrome following the use of acetyl acid by children or young people with accelerated toxic infections, in the form of severe liver dysfunction combined brain disease, which, although rare, can cause death and be used with caution。
